KANTR integral membrane proteinGenealiases: LINC01155 · Spasm
Q-omics provides the consensus-scored KANTR profile across patient tissues and cancer cell-line models. KANTR expression is associated with patient survival in 26 of 34 cancer types, with the highest sampling consensus in KICH. Among the 18 cancer types available for tumor–normal comparison, KANTR is differentially expressed in 9, with the highest sampling consensus in LIHC. Additionally, KANTR RNA expression shows 20,044 significant gene co-expression associations, with the highest sampling consensus in UVM. Together, these results highlight KICH, LIHC, and UVM as cancer lineages where KANTR shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for KANTR — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes KANTR survival associations across molecular data types. KANTR RNA expression shows survival associations in the most cancer types (26), followed by mutation status (2). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible KANTR RNA expression–survival associations across cancer types. High KANTR expression shows unfavorable associations in KICH, MESO, COAD, UVM and KIRC, but favorable associations in BLCA. The KICH Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p = .001). Together, the overview and detailed table identify KICH as the clearest survival context for KANTR RNA expression.
This table summarizes KANTR tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 9. The strongest signals are observed in LIHC for RNA.
This table ranks reproducible tumor–normal expression differences for KANTR. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. KANTR shows lower tumor expression in THCA and higher tumor expression in LIHC, BLCA, KIRC, READ and CHOL. The LIHC box plot shows higher KANTR RNA expression in tumor versus normal tissue (log2 FC = +0.217, t-test p = .001).
This table shows molecular features associated with KANTR in patient tissues and cancer cell lines. In patient samples, KANTR shows the broadest associations at the RNA and protein expression levels, with UVM recurring as the lineage with the largest associated feature set. In cancer cell lines, KANTR RNA and mutation anchors are most strongly linked to RNA-expression features, especially in UPPER_AERODIGESTIVE_TRACT.
