Q-omics provides the consensus-scored VN1R4 profile across patient tissues and cancer cell-line models. VN1R4 expression is associated with patient survival in 7 of 34 cancer types, with the highest sampling consensus in ACC. Additionally, VN1R4 RNA expression shows 5,602 significant pathway-activity associations, with the highest sampling consensus in STAD. Together, these results highlight ACC, and STAD as cancer lineages where VN1R4 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for VN1R4 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes VN1R4 survival associations across molecular data types. VN1R4 RNA expression shows survival associations in the most cancer types (7), followed by mutation status (3). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible VN1R4 RNA expression–survival associations across cancer types. High VN1R4 expression shows unfavorable associations in ACC, CESC, STAD, KIRC, MESO and SKCM. The ACC Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify ACC as the clearest survival context for VN1R4 RNA expression.
This table shows molecular features associated with VN1R4 in patient tissues and cancer cell lines. In patient samples, VN1R4 shows the broadest associations at the RNA and protein expression levels, with STAD recurring as the lineage with the largest associated feature set. In cancer cell lines, VN1R4 RNA and mutation anchors are most strongly linked to RNA-expression features, especially in CNS, while CRISPR and shRNA rows add functional-dependency signals in LUNG_NSCLC_LUAD and LARGE_INTESTINE.
