Q-omics provides the consensus-scored TRIM51HP profile across patient tissues and cancer cell-line models. TRIM51HP expression is associated with patient survival in 14 of 34 cancer types, with the highest sampling consensus in DLBC. Additionally, TRIM51HP RNA expression shows 7,585 significant gene co-expression associations, with the highest sampling consensus in TGCT. Together, these results highlight DLBC, and TGCT as cancer lineages where TRIM51HP shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for TRIM51HP — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes TRIM51HP survival associations across molecular data types. TRIM51HP RNA expression shows survival associations in the most cancer types (14), followed by mutation status (5). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible TRIM51HP RNA expression–survival associations across cancer types. High TRIM51HP expression shows unfavorable associations in DLBC, BLCA, UVM, UCS, HNSC and LIHC. The DLBC Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify DLBC as the clearest survival context for TRIM51HP RNA expression.
This table shows molecular features associated with TRIM51HP in patient tissues and cancer cell lines. In patient samples, TRIM51HP shows the broadest associations at the RNA and protein expression levels, with TGCT recurring as the lineage with the largest associated feature set. In cancer cell lines, TRIM51HP RNA and mutation anchors are most strongly linked to RNA-expression features, especially in LARGE_INTESTINE, while CRISPR and shRNA rows add functional-dependency signals in LUNG_NSCLC_LUAD.
