plaque enriched lncRNA in atherosclerotic and inflammatory bowel macrophage regulationGenealiases: GCRL1 · LINC01272 · SMIM25
Q-omics provides the consensus-scored PELATON profile across patient tissues and cancer cell-line models. PELATON expression is associated with patient survival in 23 of 34 cancer types, with the highest sampling consensus in UVM. Among the 18 cancer types available for tumor–normal comparison, PELATON is differentially expressed in 13, with the highest sampling consensus in KIRC. Additionally, PELATON RNA expression shows 22,789 significant protein co-abundance associations, with the highest sampling consensus in GBM. Together, these results highlight UVM, KIRC, and GBM as cancer lineages where PELATON shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for PELATON — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes PELATON survival associations across molecular data types. PELATON RNA expression shows survival associations in the most cancer types (23). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible PELATON RNA expression–survival associations across cancer types. High PELATON expression shows unfavorable associations in UVM, ACC, BRCA, LGG, COAD and LAML. The UVM Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify UVM as the clearest survival context for PELATON RNA expression.
This table summarizes PELATON tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 13. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for PELATON. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. PELATON shows lower tumor expression in LUAD and LUSC and higher tumor expression in KIRC, COAD, KIRP and STAD. The KIRC box plot shows higher PELATON RNA expression in tumor versus normal tissue (log2 FC = +1.852, t-test p < 0.001).
This table shows molecular features associated with PELATON in patient tissues and cancer cell lines. In patient samples, PELATON shows the broadest associations at the RNA and protein expression levels, with GBM recurring as the lineage with the largest associated feature set.
