ST13, Hsp70 interacting protein pseudogene 4Genealiases: FAM10A4 · FAM10A4P
Q-omics provides the consensus-scored ST13P4 profile across patient tissues and cancer cell-line models. ST13P4 expression is associated with patient survival in 23 of 34 cancer types, with the highest sampling consensus in KIRC. Among the 18 cancer types available for tumor–normal comparison, ST13P4 is differentially expressed in 8, with the highest sampling consensus in KIRP. Additionally, ST13P4 RNA expression shows 17,390 significant gene co-expression associations, with the highest sampling consensus in ACC. Together, these results highlight KIRC, KIRP, and ACC as cancer lineages where ST13P4 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for ST13P4 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes ST13P4 survival associations across molecular data types. ST13P4 RNA expression shows survival associations in the most cancer types (23), followed by mutation status (4). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible ST13P4 RNA expression–survival associations across cancer types. High ST13P4 expression shows unfavorable associations in LUAD, ACC and LIHC, but favorable associations in KIRC, THCA and UCS. The KIRC Kaplan–Meier curve shows clear separation, with the low-expression group declining faster, consistent with the favorable association (log-rank p < 0.001). Together, the overview and detailed table identify KIRC as the clearest survival context for ST13P4 RNA expression.
This table summarizes ST13P4 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 8. The strongest signals are observed in KIRP for RNA.
This table ranks reproducible tumor–normal expression differences for ST13P4. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. ST13P4 shows lower tumor expression in KIRP and BRCA and higher tumor expression in HNSC, KIRC, CHOL and LUSC. The KIRP box plot shows higher ST13P4 RNA expression in normal versus tumor tissue (log2 FC = −0.293, t-test p < 0.001).
This table shows molecular features associated with ST13P4 in patient tissues and cancer cell lines. In patient samples, ST13P4 shows the broadest associations at the RNA and protein expression levels, with ACC recurring as the lineage with the largest associated feature set. In cancer cell lines, ST13P4 RNA and mutation anchors are most strongly linked to RNA-expression features, especially in OVARY, while CRISPR and shRNA rows add functional-dependency signals in STOMACH.
