ribosomal protein S23 pseudogene 10Genealiases: []
Q-omics provides the consensus-scored RPS23P10 profile across patient tissues and cancer cell-line models. RPS23P10 expression is associated with patient survival in 12 of 34 cancer types, with the highest sampling consensus in KIRC. Among the 18 cancer types available for tumor–normal comparison, RPS23P10 is differentially expressed in 5, with the highest sampling consensus in PAAD. Additionally, RPS23P10 RNA expression shows 6,523 significant pathway-activity associations, with the highest sampling consensus in STAD. Together, these results highlight KIRC, PAAD, and STAD as cancer lineages where RPS23P10 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for RPS23P10 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes RPS23P10 survival associations across molecular data types. RPS23P10 RNA expression shows survival associations in the most cancer types (12). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible RPS23P10 RNA expression–survival associations across cancer types. High RPS23P10 expression shows unfavorable associations in KIRC, TGCT, UCEC, DLBC, MESO and ACC. The KIRC Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p = .007). Together, the overview and detailed table identify KIRC as the clearest survival context for RPS23P10 RNA expression.
This table summarizes RPS23P10 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 5. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for RPS23P10. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. RPS23P10 shows lower tumor expression in PAAD, LUSC and LUAD and higher tumor expression in COAD and KIRC. The PAAD box plot shows higher RPS23P10 RNA expression in normal versus tumor tissue (log2 FC = −0.195, t-test p = .049).
This table shows molecular features associated with RPS23P10 in patient tissues and cancer cell lines. In patient samples, RPS23P10 shows the broadest associations at the RNA and protein expression levels, with STAD recurring as the lineage with the largest associated feature set.
