Q-omics provides the consensus-scored RASSF10-DT profile across patient tissues and cancer cell-line models. RASSF10-DT expression is associated with patient survival in 22 of 34 cancer types, with the highest sampling consensus in KIRC. Among the 18 cancer types available for tumor–normal comparison, RASSF10-DT is differentially expressed in 13, with the highest sampling consensus in KIRC. Additionally, RASSF10-DT RNA expression shows 15,665 significant gene co-expression associations, with the highest sampling consensus in THYM. Together, these results highlight KIRC, and THYM as cancer lineages where RASSF10-DT shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for RASSF10-DT — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes RASSF10-DT survival associations across molecular data types. RASSF10-DT RNA expression shows survival associations in the most cancer types (22). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible RASSF10-DT RNA expression–survival associations across cancer types. High RASSF10-DT expression shows unfavorable associations in LGG, UCEC and LUSC, but favorable associations in KIRC, KIRP and HNSC. The KIRC Kaplan–Meier curve shows clear separation, with the low-expression group declining faster, consistent with the favorable association (log-rank p < 0.001). Together, the overview and detailed table identify KIRC as the clearest survival context for RASSF10-DT RNA expression.
This table summarizes RASSF10-DT tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 13. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for RASSF10-DT. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. RASSF10-DT shows lower tumor expression in KIRC, KIRP and THCA and higher tumor expression in COAD, BLCA and LUSC. The KIRC box plot shows higher RASSF10-DT RNA expression in normal versus tumor tissue (log2 FC = −1.089, t-test p < 0.001).
This table shows molecular features associated with RASSF10-DT in patient tissues and cancer cell lines. In patient samples, RASSF10-DT shows the broadest associations at the RNA and protein expression levels, with THYM recurring as the lineage with the largest associated feature set.
