Q-omics provides the consensus-scored MNX1-AS2 profile across patient tissues and cancer cell-line models. MNX1-AS2 expression is associated with patient survival in 22 of 34 cancer types, with the highest sampling consensus in KIRC. Among the 18 cancer types available for tumor–normal comparison, MNX1-AS2 is differentially expressed in 14, with the highest sampling consensus in KIRP. Additionally, MNX1-AS2 RNA expression shows 12,652 significant gene co-expression associations, with the highest sampling consensus in ESCA. Together, these results highlight KIRC, KIRP, and ESCA as cancer lineages where MNX1-AS2 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for MNX1-AS2 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes MNX1-AS2 survival associations across molecular data types. MNX1-AS2 RNA expression shows survival associations in the most cancer types (22). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible MNX1-AS2 RNA expression–survival associations across cancer types. High MNX1-AS2 expression shows unfavorable associations in KIRC, DLBC, ACC, KIRP, KICH and MESO. The KIRC Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify KIRC as the clearest survival context for MNX1-AS2 RNA expression.
This table summarizes MNX1-AS2 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 14. The strongest signals are observed in KIRP for RNA.
This table ranks reproducible tumor–normal expression differences for MNX1-AS2. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. MNX1-AS2 shows higher tumor expression in KIRP, BLCA, STAD, LUAD, COAD and KIRC. The KIRP box plot shows higher MNX1-AS2 RNA expression in tumor versus normal tissue (log2 FC = +0.860, t-test p < 0.001).
This table shows molecular features associated with MNX1-AS2 in patient tissues and cancer cell lines. In patient samples, MNX1-AS2 shows the broadest associations at the RNA and protein expression levels, with ESCA recurring as the lineage with the largest associated feature set.
