Q-omics provides the consensus-scored CIAPIN1P profile across patient tissues and cancer cell-line models. CIAPIN1P expression is associated with patient survival in 21 of 34 cancer types, with the highest sampling consensus in CESC. Among the 18 cancer types available for tumor–normal comparison, CIAPIN1P is differentially expressed in 7, with the highest sampling consensus in COAD. Additionally, CIAPIN1P RNA expression shows 14,521 significant gene co-expression associations, with the highest sampling consensus in THYM. Together, these results highlight CESC, COAD, and THYM as cancer lineages where CIAPIN1P shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for CIAPIN1P — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes CIAPIN1P survival associations across molecular data types. CIAPIN1P RNA expression shows survival associations in the most cancer types (21). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible CIAPIN1P RNA expression–survival associations across cancer types. High CIAPIN1P expression shows unfavorable associations in STAD and UCS, but favorable associations in CESC, KIRC, LAML and BRCA. The CESC Kaplan–Meier curve shows clear separation, with the low-expression group declining faster, consistent with the favorable association (log-rank p = .008). Together, the overview and detailed table identify CESC as the clearest survival context for CIAPIN1P RNA expression.
This table summarizes CIAPIN1P tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 7. The strongest signals are observed in COAD for RNA.
This table ranks reproducible tumor–normal expression differences for CIAPIN1P. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. CIAPIN1P shows lower tumor expression in KICH, THCA and KIRC and higher tumor expression in COAD, PRAD and LUAD. The COAD box plot shows higher CIAPIN1P RNA expression in tumor versus normal tissue (log2 FC = +0.440, t-test p < 0.001).
This table shows molecular features associated with CIAPIN1P in patient tissues and cancer cell lines. In patient samples, CIAPIN1P shows the broadest associations at the RNA and protein expression levels, with THYM recurring as the lineage with the largest associated feature set.
