BCL2 interacting protein 3 pseudogene 6Genealiases: []
Q-omics provides the consensus-scored BNIP3P6 profile across patient tissues and cancer cell-line models. BNIP3P6 expression is associated with patient survival in 10 of 34 cancer types, with the highest sampling consensus in READ. Among the 18 cancer types available for tumor–normal comparison, BNIP3P6 is differentially expressed in 1, with the highest sampling consensus in LUAD. Additionally, BNIP3P6 RNA expression shows 2,154 significant mutation-linked associations, with the highest sampling consensus in UCEC. Together, these results highlight READ, LUAD, and UCEC as cancer lineages where BNIP3P6 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for BNIP3P6 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes BNIP3P6 survival associations across molecular data types. BNIP3P6 RNA expression shows survival associations in the most cancer types (10). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible BNIP3P6 RNA expression–survival associations across cancer types. High BNIP3P6 expression shows unfavorable associations in READ, KIRC, LUAD, STAD, LAML and COAD. The READ Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify READ as the clearest survival context for BNIP3P6 RNA expression.
This table summarizes BNIP3P6 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 1. The strongest signals are observed in LUAD for RNA.
This table ranks reproducible tumor–normal expression differences for BNIP3P6. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. BNIP3P6 shows higher tumor expression in LUAD. The LUAD box plot shows higher BNIP3P6 RNA expression in tumor versus normal tissue (log2 FC = +0.072, t-test p = .021).
This table shows molecular features associated with BNIP3P6 in patient tissues and cancer cell lines. In patient samples, BNIP3P6 shows the broadest associations at the RNA and protein expression levels, with UCEC recurring as the lineage with the largest associated feature set.
