BCL2 interacting protein 3 pseudogene 5Genealiases: []
Q-omics provides the consensus-scored BNIP3P5 profile across patient tissues and cancer cell-line models. BNIP3P5 expression is associated with patient survival in 24 of 34 cancer types, with the highest sampling consensus in CESC. Among the 18 cancer types available for tumor–normal comparison, BNIP3P5 is differentially expressed in 7, with the highest sampling consensus in LUAD. Additionally, BNIP3P5 RNA expression shows 18,879 significant gene co-expression associations, with the highest sampling consensus in THYM. Together, these results highlight CESC, LUAD, and THYM as cancer lineages where BNIP3P5 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for BNIP3P5 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes BNIP3P5 survival associations across molecular data types. BNIP3P5 RNA expression shows survival associations in the most cancer types (24). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible BNIP3P5 RNA expression–survival associations across cancer types. High BNIP3P5 expression shows unfavorable associations in CESC, LGG, UCEC, KIRP and MESO, but favorable associations in LUAD. The CESC Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify CESC as the clearest survival context for BNIP3P5 RNA expression.
This table summarizes BNIP3P5 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 7. The strongest signals are observed in LUAD for RNA.
This table ranks reproducible tumor–normal expression differences for BNIP3P5. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. BNIP3P5 shows lower tumor expression in LUAD and LUSC and higher tumor expression in HNSC, STAD, LIHC and THCA. The LUAD box plot shows higher BNIP3P5 RNA expression in normal versus tumor tissue (log2 FC = −1.100, t-test p < 0.001).
This table shows molecular features associated with BNIP3P5 in patient tissues and cancer cell lines. In patient samples, BNIP3P5 shows the broadest associations at the RNA and protein expression levels, with THYM recurring as the lineage with the largest associated feature set.
