ATP synthase membrane subunit f pseudogene 3Genealiases: ATP5J2P3 · bA159H20.5
Q-omics provides the consensus-scored ATP5MFP3 profile across patient tissues and cancer cell-line models. ATP5MFP3 expression is associated with patient survival in 12 of 34 cancer types, with the highest sampling consensus in SKCM. Among the 18 cancer types available for tumor–normal comparison, ATP5MFP3 is differentially expressed in 2, with the highest sampling consensus in KIRC. Additionally, ATP5MFP3 RNA expression shows 5,925 significant pathway-activity associations, with the highest sampling consensus in STAD. Together, these results highlight SKCM, KIRC, and STAD as cancer lineages where ATP5MFP3 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for ATP5MFP3 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes ATP5MFP3 survival associations across molecular data types. ATP5MFP3 RNA expression shows survival associations in the most cancer types (12). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible ATP5MFP3 RNA expression–survival associations across cancer types. High ATP5MFP3 expression shows unfavorable associations in SKCM, KIRP, READ, ACC and LUAD, but favorable associations in ESCA. The SKCM Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify SKCM as the clearest survival context for ATP5MFP3 RNA expression.
This table summarizes ATP5MFP3 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 2. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for ATP5MFP3. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. ATP5MFP3 shows higher tumor expression in KIRC and KIRP. The KIRC box plot shows higher ATP5MFP3 RNA expression in tumor versus normal tissue (log2 FC = +0.191, t-test p < 0.001).
This table shows molecular features associated with ATP5MFP3 in patient tissues and cancer cell lines. In patient samples, ATP5MFP3 shows the broadest associations at the RNA and protein expression levels, with STAD recurring as the lineage with the largest associated feature set.
