TMSB10P1

Across TCGA pan-cancer cohorts, TMSB10P1 RNA differs between tumor and matched normal tissue in 14 of 18 cancer types tested, making tumor–normal expression one of TMSB10P1’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where TMSB10P1 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types TMSB10P1 is over-expressed in tumor.

KIRC, COAD, and HNSC are the cancer types where TMSB10P1 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in TMSB10P1 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	Male	All	+1.477	<.001	12	view →
COAD	Female	II,III,IV	+2.205	<.001	10	view →
HNSC	Male	All	+1.317	<.001	8	view →
KIRP	Male	All	+1.309	<.001	8	view →
LIHC	Female	II,III,IV	+0.979	<.001	8	view →
THCA	All	II,III,IV	+0.856	.001	8	view →
UCEC	All	All	+1.284	<.001	6	view →
LUAD	All	All	+0.817	<.001	5	view →
STAD	All	All	+0.567	.010	4	view →
BRCA	All	All	+0.549	<.001	4	view →
CHOL	All	All	+1.396	<.001	3	view →
ESCA	Male	All	+1.214	.027	2	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 12 strongest of 14 lineages.

TMSB10P1–KIRC

Open the KIRC breakdown →