Q-omics provides the consensus-scored TMSB10P1 profile across patient tissues and cancer cell-line models. TMSB10P1 expression is associated with patient survival in 24 of 34 cancer types, with the highest sampling consensus in STAD. Among the 18 cancer types available for tumor–normal comparison, TMSB10P1 is differentially expressed in 14, with the highest sampling consensus in KIRC. Additionally, TMSB10P1 RNA expression shows 15,875 significant gene co-expression associations, with the highest sampling consensus in THYM. Together, these results highlight STAD, KIRC, and THYM as cancer lineages where TMSB10P1 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for TMSB10P1 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes TMSB10P1 survival associations across molecular data types. TMSB10P1 RNA expression shows survival associations in the most cancer types (24). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible TMSB10P1 RNA expression–survival associations across cancer types. High TMSB10P1 expression shows unfavorable associations in STAD, UVM, LGG, LUAD and ACC, but favorable associations in THCA. The STAD Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p = .001). Together, the overview and detailed table identify STAD as the clearest survival context for TMSB10P1 RNA expression.
This table summarizes TMSB10P1 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 14. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for TMSB10P1. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. TMSB10P1 shows higher tumor expression in KIRC, COAD, HNSC, KIRP, LIHC and THCA. The KIRC box plot shows higher TMSB10P1 RNA expression in tumor versus normal tissue (log2 FC = +1.477, t-test p < 0.001).
This table shows molecular features associated with TMSB10P1 in patient tissues and cancer cell lines. In patient samples, TMSB10P1 shows the broadest associations at the RNA and protein expression levels, with THYM recurring as the lineage with the largest associated feature set.
