Q-omics provides the consensus-scored TCF3P1 profile across patient tissues and cancer cell-line models. TCF3P1 expression is associated with patient survival in 20 of 34 cancer types, with the highest sampling consensus in ACC. Among the 18 cancer types available for tumor–normal comparison, TCF3P1 is differentially expressed in 7, with the highest sampling consensus in COAD. Additionally, TCF3P1 RNA expression shows 15,446 significant gene co-expression associations, with the highest sampling consensus in DLBC. Together, these results highlight ACC, COAD, and DLBC as cancer lineages where TCF3P1 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for TCF3P1 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes TCF3P1 survival associations across molecular data types. TCF3P1 RNA expression shows survival associations in the most cancer types (20). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible TCF3P1 RNA expression–survival associations across cancer types. High TCF3P1 expression shows unfavorable associations in ACC, UVM and LUSC, but favorable associations in SKCM, HNSC and BRCA. The ACC Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify ACC as the clearest survival context for TCF3P1 RNA expression.
This table summarizes TCF3P1 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 7. The strongest signals are observed in THCA for RNA.
This table ranks reproducible tumor–normal expression differences for TCF3P1. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. TCF3P1 shows lower tumor expression in THCA and KICH and higher tumor expression in COAD, STAD, KIRC and LIHC. The COAD box plot shows higher TCF3P1 RNA expression in tumor versus normal tissue (log2 FC = +0.104, t-test p < 0.001).
This table shows molecular features associated with TCF3P1 in patient tissues and cancer cell lines. In patient samples, TCF3P1 shows the broadest associations at the RNA and protein expression levels, with DLBC recurring as the lineage with the largest associated feature set.
