SRP68P3

Across TCGA pan-cancer cohorts, SRP68P3 RNA differs between tumor and matched normal tissue in 5 of 18 cancer types tested, making tumor–normal expression one of SRP68P3’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where SRP68P3 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types SRP68P3 is over-expressed in tumor, although a few such as THCA show the opposite, repressed pattern.

KIRC, THCA, and CHOL are the cancer types where SRP68P3 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in SRP68P3 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	Male	III,IV	+0.055	.009	5	view →
THCA	Female	II,III,IV	−0.099	.017	3	view →
CHOL	All	All	+0.223	.038	1	view →
KICH	All	II,III,IV	+0.083	.033	1	view →
LUAD	All	All	+0.052	.037	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 5 strongest of 5 lineages.

SRP68P3–KIRC

Open the KIRC breakdown →