RPL36A-HNRNPH2

Across TCGA pan-cancer cohorts, RPL36A-HNRNPH2 RNA differs between tumor and matched normal tissue in 11 of 18 cancer types tested, making tumor–normal expression one of RPL36A-HNRNPH2’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where RPL36A-HNRNPH2 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types RPL36A-HNRNPH2 is over-expressed in tumor, although a few such as UCEC and KICH show the opposite, repressed pattern.

KIRC, LIHC, and UCEC are the cancer types where RPL36A-HNRNPH2 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in RPL36A-HNRNPH2 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	Male	All	+0.206	<.001	7	view →
LIHC	All	All	+0.120	<.001	6	view →
UCEC	All	IV	−0.255	.003	4	view →
LUSC	Male	All	+0.184	<.001	4	view →
LUAD	Male	III,IV	+0.271	.034	3	view →
KIRP	All	III,IV	+0.180	.033	3	view →
READ	All	All	+0.348	.007	2	view →
KICH	Male	III,IV	−0.237	.006	2	view →
PRAD	All	All	+0.175	<.001	2	view →
COAD	All	All	+0.154	.015	2	view →
STAD	All	All	+0.204	.041	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 11 strongest of 11 lineages.

RPL36A-HNRNPH2–KIRC

Open the KIRC breakdown →