Q-omics provides the consensus-scored RN7SL268P profile across patient tissues and cancer cell-line models. RN7SL268P expression is associated with patient survival in 22 of 34 cancer types, with the highest sampling consensus in KIRP. Among the 18 cancer types available for tumor–normal comparison, RN7SL268P is differentially expressed in 12, with the highest sampling consensus in KIRC. Additionally, RN7SL268P RNA expression shows 19,479 significant gene co-expression associations, with the highest sampling consensus in KIRP. Together, these results highlight KIRP, and KIRC as cancer lineages where RN7SL268P shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for RN7SL268P — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes RN7SL268P survival associations across molecular data types. RN7SL268P RNA expression shows survival associations in the most cancer types (22). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible RN7SL268P RNA expression–survival associations across cancer types. High RN7SL268P expression shows unfavorable associations in COAD, but favorable associations in KIRP, READ, PAAD, LUAD and ACC. The KIRP Kaplan–Meier curve shows clear separation, with the low-expression group declining faster, consistent with the favorable association (log-rank p = .001). Together, the overview and detailed table identify KIRP as the clearest survival context for RN7SL268P RNA expression.
This table summarizes RN7SL268P tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 12. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for RN7SL268P. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. RN7SL268P shows lower tumor expression in KIRC, KIRP and THCA and higher tumor expression in CHOL, BRCA and UCEC. The KIRC box plot shows higher RN7SL268P RNA expression in normal versus tumor tissue (log2 FC = −0.890, t-test p < 0.001).
This table shows molecular features associated with RN7SL268P in patient tissues and cancer cell lines. In patient samples, RN7SL268P shows the broadest associations at the RNA and protein expression levels, with KIRP recurring as the lineage with the largest associated feature set.
