Q-omics provides the consensus-scored RN7SL248P profile across patient tissues and cancer cell-line models. RN7SL248P expression is associated with patient survival in 7 of 34 cancer types, with the highest sampling consensus in CESC. Among the 18 cancer types available for tumor–normal comparison, RN7SL248P is differentially expressed in 3, with the highest sampling consensus in KIRC. Additionally, RN7SL248P RNA expression shows 5,965 significant pathway-activity associations, with the highest sampling consensus in STAD. Together, these results highlight CESC, KIRC, and STAD as cancer lineages where RN7SL248P shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for RN7SL248P — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes RN7SL248P survival associations across molecular data types. RN7SL248P RNA expression shows survival associations in the most cancer types (7), followed by mutation status (4). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible RN7SL248P RNA expression–survival associations across cancer types. High RN7SL248P expression shows unfavorable associations in CESC, LUAD, ESCA, SKCM and OV, but favorable associations in STAD. The CESC Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify CESC as the clearest survival context for RN7SL248P RNA expression.
This table summarizes RN7SL248P tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 3. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for RN7SL248P. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. RN7SL248P shows lower tumor expression in STAD and higher tumor expression in KIRC and KIRP. The KIRC box plot shows higher RN7SL248P RNA expression in tumor versus normal tissue (log2 FC = +0.074, t-test p < 0.001).
This table shows molecular features associated with RN7SL248P in patient tissues and cancer cell lines. In patient samples, RN7SL248P shows the broadest associations at the RNA and protein expression levels, with STAD recurring as the lineage with the largest associated feature set.
