RN7SL213P

Across TCGA pan-cancer cohorts, RN7SL213P RNA differs between tumor and matched normal tissue in 8 of 18 cancer types tested, making tumor–normal expression one of RN7SL213P’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal papillary cell carcinoma (KIRP), where RN7SL213P RNA is more highly expressed in tumor relative to normal tissue. In most cancer types RN7SL213P is over-expressed in tumor.

KIRP, CHOL, and KIRC are the cancer types where RN7SL213P tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in RN7SL213P RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRP	Female	II,III,IV	+0.258	<.001	5	view →
CHOL	All	All	+0.503	.006	4	view →
KIRC	All	All	+0.060	.013	3	view →
UCEC	All	IV	+0.560	.011	2	view →
BRCA	All	All	+0.111	.022	2	view →
HNSC	All	All	+0.050	.026	2	view →
STAD	Male	II,III,IV	+0.145	.041	1	view →
LIHC	All	II,III,IV	+0.046	.047	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 8 strongest of 8 lineages.

RN7SL213P–KIRP

Open the KIRP breakdown →