RDH10-AS1

Across TCGA pan-cancer cohorts, RDH10-AS1 RNA differs between tumor and matched normal tissue in 11 of 18 cancer types tested, making tumor–normal expression one of RDH10-AS1’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where RDH10-AS1 RNA is repressed in tumor relative to normal tissue. In most cancer types RDH10-AS1 is over-expressed in tumor, although a few such as KIRC and KIRP show the opposite, repressed pattern.

KIRC, UCEC, and COAD are the cancer types where RDH10-AS1 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in RDH10-AS1 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	Female	All	−0.503	<.001	11	view →
UCEC	All	II,III,IV	+0.690	<.001	6	view →
COAD	All	II,III,IV	+0.112	.001	5	view →
LIHC	All	All	+0.093	<.001	5	view →
CHOL	All	All	+0.462	<.001	4	view →
LUAD	All	IV	+0.500	.028	3	view →
KIRP	All	All	−0.434	.002	2	view →
KICH	All	All	−0.289	.008	2	view →
THCA	Male	IV	+0.073	.005	2	view →
READ	All	All	+0.287	.040	1	view →
STAD	Male	II,III,IV	+0.265	.040	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 11 strongest of 11 lineages.

RDH10-AS1–KIRC

Open the KIRC breakdown →