Q-omics provides the consensus-scored RAB11B-AS1 profile across patient tissues and cancer cell-line models. RAB11B-AS1 expression is associated with patient survival in 27 of 34 cancer types, with the highest sampling consensus in UCEC. Among the 18 cancer types available for tumor–normal comparison, RAB11B-AS1 is differentially expressed in 12, with the highest sampling consensus in LUAD. Additionally, RAB11B-AS1 RNA expression shows 17,969 significant gene co-expression associations, with the highest sampling consensus in THYM. Together, these results highlight UCEC, LUAD, and THYM as cancer lineages where RAB11B-AS1 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for RAB11B-AS1 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes RAB11B-AS1 survival associations across molecular data types. RAB11B-AS1 RNA expression shows survival associations in the most cancer types (27). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible RAB11B-AS1 RNA expression–survival associations across cancer types. High RAB11B-AS1 expression shows unfavorable associations in UVM, LGG and KICH, but favorable associations in UCEC, CESC and HNSC. The UCEC Kaplan–Meier curve shows clear separation, with the low-expression group declining faster, consistent with the favorable association (log-rank p < 0.001). Together, the overview and detailed table identify UCEC as the clearest survival context for RAB11B-AS1 RNA expression.
This table summarizes RAB11B-AS1 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 12. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for RAB11B-AS1. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. RAB11B-AS1 shows lower tumor expression in LUAD, BRCA, HNSC, STAD and UCEC and higher tumor expression in KIRC. The LUAD box plot shows higher RAB11B-AS1 RNA expression in normal versus tumor tissue (log2 FC = −0.678, t-test p < 0.001).
This table shows molecular features associated with RAB11B-AS1 in patient tissues and cancer cell lines. In patient samples, RAB11B-AS1 shows the broadest associations at the RNA and protein expression levels, with THYM recurring as the lineage with the largest associated feature set.
