peptidylprolyl isomerase A pseudogene 32Genealiases: []
Q-omics provides the consensus-scored PPIAP32 profile across patient tissues and cancer cell-line models. PPIAP32 expression is associated with patient survival in 19 of 34 cancer types, with the highest sampling consensus in THCA. Among the 18 cancer types available for tumor–normal comparison, PPIAP32 is differentially expressed in 4, with the highest sampling consensus in HNSC. Additionally, PPIAP32 RNA expression shows 7,502 significant gene co-expression associations, with the highest sampling consensus in UVM. Together, these results highlight THCA, HNSC, and UVM as cancer lineages where PPIAP32 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for PPIAP32 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes PPIAP32 survival associations across molecular data types. PPIAP32 RNA expression shows survival associations in the most cancer types (19). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible PPIAP32 RNA expression–survival associations across cancer types. High PPIAP32 expression shows unfavorable associations in THCA, TGCT, MESO and THYM, but favorable associations in UCS and BRCA. The THCA Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify THCA as the clearest survival context for PPIAP32 RNA expression.
This table summarizes PPIAP32 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 4. The strongest signals are observed in HNSC for RNA.
This table ranks reproducible tumor–normal expression differences for PPIAP32. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. PPIAP32 shows higher tumor expression in HNSC, LIHC, KIRC and LUAD. The HNSC box plot shows higher PPIAP32 RNA expression in tumor versus normal tissue (log2 FC = +0.035, t-test p = .015).
This table shows molecular features associated with PPIAP32 in patient tissues and cancer cell lines. In patient samples, PPIAP32 shows the broadest associations at the RNA and protein expression levels, with UVM recurring as the lineage with the largest associated feature set.
