Q-omics provides the consensus-scored MIR3200 profile across patient tissues and cancer cell-line models. MIR3200 expression is associated with patient survival in 17 of 34 cancer types, with the highest sampling consensus in MESO. Among the 18 cancer types available for tumor–normal comparison, MIR3200 is differentially expressed in 1, with the highest sampling consensus in THCA. Additionally, MIR3200 RNA expression shows 5,880 significant pathway-activity associations, with the highest sampling consensus in STAD. Together, these results highlight MESO, THCA, and STAD as cancer lineages where MIR3200 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for MIR3200 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes MIR3200 survival associations across molecular data types. MIR3200 RNA expression shows survival associations in the most cancer types (17). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible MIR3200 RNA expression–survival associations across cancer types. High MIR3200 expression shows unfavorable associations in MESO, KIRP, LIHC, LUAD, GBM and BRCA. The MESO Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify MESO as the clearest survival context for MIR3200 RNA expression.
This table summarizes MIR3200 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 1. The strongest signals are observed in THCA for RNA.
This table ranks reproducible tumor–normal expression differences for MIR3200. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. MIR3200 shows lower tumor expression in THCA. The THCA box plot shows higher MIR3200 RNA expression in normal versus tumor tissue (log2 FC = −0.075, t-test p = .028).
This table shows molecular features associated with MIR3200 in patient tissues and cancer cell lines. In patient samples, MIR3200 shows the broadest associations at the RNA and protein expression levels, with STAD recurring as the lineage with the largest associated feature set.
