Q-omics provides the consensus-scored MIR210HG profile across patient tissues and cancer cell-line models. MIR210HG expression is associated with patient survival in 25 of 34 cancer types, with the highest sampling consensus in KICH. Among the 18 cancer types available for tumor–normal comparison, MIR210HG is differentially expressed in 15, with the highest sampling consensus in KIRC. Additionally, MIR210HG RNA expression shows 15,870 significant gene co-expression associations, with the highest sampling consensus in ACC. Together, these results highlight KICH, KIRC, and ACC as cancer lineages where MIR210HG shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for MIR210HG — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes MIR210HG survival associations across molecular data types. MIR210HG RNA expression shows survival associations in the most cancer types (25). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible MIR210HG RNA expression–survival associations across cancer types. High MIR210HG expression shows unfavorable associations in KICH, LIHC, ACC, COAD and LUAD, but favorable associations in DLBC. The KICH Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify KICH as the clearest survival context for MIR210HG RNA expression.
This table summarizes MIR210HG tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 15. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for MIR210HG. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. MIR210HG shows lower tumor expression in THCA and higher tumor expression in KIRC, LUSC, KIRP, UCEC and BLCA. The KIRC box plot shows higher MIR210HG RNA expression in tumor versus normal tissue (log2 FC = +2.750, t-test p < 0.001).
This table shows molecular features associated with MIR210HG in patient tissues and cancer cell lines. In patient samples, MIR210HG shows the broadest associations at the RNA and protein expression levels, with ACC recurring as the lineage with the largest associated feature set.
