Q-omics provides the consensus-scored MIR137HG profile across patient tissues and cancer cell-line models. MIR137HG expression is associated with patient survival in 18 of 34 cancer types, with the highest sampling consensus in KIRP. Among the 18 cancer types available for tumor–normal comparison, MIR137HG is differentially expressed in 9, with the highest sampling consensus in COAD. Additionally, MIR137HG RNA expression shows 12,291 significant gene co-expression associations, with the highest sampling consensus in PCPG. Together, these results highlight KIRP, COAD, and PCPG as cancer lineages where MIR137HG shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for MIR137HG — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes MIR137HG survival associations across molecular data types. MIR137HG RNA expression shows survival associations in the most cancer types (18), followed by mutation status (1). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible MIR137HG RNA expression–survival associations across cancer types. High MIR137HG expression shows unfavorable associations in KIRP, LIHC, HNSC, KIRC and BRCA, but favorable associations in ACC. The KIRP Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify KIRP as the clearest survival context for MIR137HG RNA expression.
This table summarizes MIR137HG tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 9. The strongest signals are observed in COAD for RNA.
This table ranks reproducible tumor–normal expression differences for MIR137HG. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. MIR137HG shows lower tumor expression in COAD, KIRC and KICH and higher tumor expression in HNSC, LUAD and LUSC. The COAD box plot shows higher MIR137HG RNA expression in normal versus tumor tissue (log2 FC = −0.041, t-test p < 0.001).
This table shows molecular features associated with MIR137HG in patient tissues and cancer cell lines. In patient samples, MIR137HG shows the broadest associations at the RNA and protein expression levels, with PCPG recurring as the lineage with the largest associated feature set.
