long intergenic non-protein coding RNA 2188Genealiases: []
Q-omics provides the consensus-scored LINC02188 profile across patient tissues and cancer cell-line models. LINC02188 expression is associated with patient survival in 21 of 34 cancer types, with the highest sampling consensus in KIRC. Among the 18 cancer types available for tumor–normal comparison, LINC02188 is differentially expressed in 10, with the highest sampling consensus in KIRC. Additionally, LINC02188 RNA expression shows 14,153 significant gene co-expression associations, with the highest sampling consensus in KIRP. Together, these results highlight KIRC, and KIRP as cancer lineages where LINC02188 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for LINC02188 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes LINC02188 survival associations across molecular data types. LINC02188 RNA expression shows survival associations in the most cancer types (21). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible LINC02188 RNA expression–survival associations across cancer types. High LINC02188 expression shows unfavorable associations in UVM, LIHC, COAD and CESC, but favorable associations in KIRC and HNSC. The KIRC Kaplan–Meier curve shows clear separation, with the low-expression group declining faster, consistent with the favorable association (log-rank p < 0.001). Together, the overview and detailed table identify KIRC as the clearest survival context for LINC02188 RNA expression.
This table summarizes LINC02188 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 10. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for LINC02188. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. LINC02188 shows lower tumor expression in BRCA and KICH and higher tumor expression in KIRC, KIRP, COAD and THCA. The KIRC box plot shows higher LINC02188 RNA expression in tumor versus normal tissue (log2 FC = +3.019, t-test p < 0.001).
This table shows molecular features associated with LINC02188 in patient tissues and cancer cell lines. In patient samples, LINC02188 shows the broadest associations at the RNA and protein expression levels, with KIRP recurring as the lineage with the largest associated feature set.
