Q-omics provides the consensus-scored LINC01611 profile across patient tissues and cancer cell-line models. LINC01611 expression is associated with patient survival in 23 of 34 cancer types, with the highest sampling consensus in KIRP. Among the 18 cancer types available for tumor–normal comparison, LINC01611 is differentially expressed in 7, with the highest sampling consensus in KIRC. Additionally, LINC01611 RNA expression shows 7,264 significant gene co-expression associations, with the highest sampling consensus in UVM. Together, these results highlight KIRP, KIRC, and UVM as cancer lineages where LINC01611 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for LINC01611 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes LINC01611 survival associations across molecular data types. LINC01611 RNA expression shows survival associations in the most cancer types (23). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible LINC01611 RNA expression–survival associations across cancer types. High LINC01611 expression shows unfavorable associations in KIRP, KIRC, ACC, UCEC, UVM and HNSC. The KIRP Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify KIRP as the clearest survival context for LINC01611 RNA expression.
This table summarizes LINC01611 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 7. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for LINC01611. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. LINC01611 shows higher tumor expression in KIRC, LUSC, LIHC, LUAD, HNSC and STAD. The KIRC box plot shows higher LINC01611 RNA expression in tumor versus normal tissue (log2 FC = +0.477, t-test p < 0.001).
This table shows molecular features associated with LINC01611 in patient tissues and cancer cell lines. In patient samples, LINC01611 shows the broadest associations at the RNA and protein expression levels, with UVM recurring as the lineage with the largest associated feature set.
