LINC00869

Mutation & survival
SurvivalMutationKaplan–Meier · TCGA cohorts

Across TCGA pan-cancer cohorts, LINC00869 Mutation is linked to patient survival in 4 of 34 cancer types, making it a survival-associated LINC00869 data layer compared with 24 for mass-spec protein.

The strongest signal is observed in ovarian serous cystadenocarcinoma (OV), where higher LINC00869 Mutation is associated with worse disease-free survival. In most high-consensus cancer types, elevated LINC00869 expression acts as an unfavorable survival marker.

OV, COAD, and UCEC are the cancer types where LINC00869 Mutation most reproducibly stratifies survival.

Mutation survival associations by lineage

Ranked by sampling consensus. AUC1 and AUC2 indicate survival in the high- and low-expression groups, respectively; the lower AUC marks the poorer-surviving group. p-values are from the log-rank test.
LineageMeasureSplitStageAUC1
high
AUC2
low
pSampling consensus
OVDFSMedianII,III,IV0.2220.541.03818view →
COADOSMedianIII,IV0.1590.692.02112view →
UCECOSMedianIV0.2310.592.0366view →
SKCMOSMedianIV0.4150.861.0146view →
Pink = unfavorable, green = favorable. Showing the 4 strongest of 4 lineages.

LINC00869–OV (DFS)

Kaplan–Meier survival curve for LINC00869 mutant vs wild-type samples in OV.

Open the OV breakdown →

Exploration