Across TCGA pan-cancer cohorts, KRTAP9-6 RNA differs between tumor and matched normal tissue in 3 of 18 cancer types tested, making tumor–normal expression one of KRTAP9-6’s most consistent transcriptional readouts.

The strongest signal is observed in stomach adenocarcinoma (STAD), where KRTAP9-6 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types KRTAP9-6 is over-expressed in tumor, although a few such as PRAD and KIRP show the opposite, repressed pattern.

STAD, PRAD, and KIRP are the cancer types where KRTAP9-6 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in KRTAP9-6 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
STAD	Male	II,III,IV	+0.023	.030	2	view →
PRAD	All	All	−0.012	.040	2	view →
KIRP	All	III,IV	−0.008	.033	2	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 3 strongest of 3 lineages.

Exploration

Same gene, other analysis: KRTAP9-6 RNA expression & survival · all KRTAP9-6 data types