Q-omics provides the consensus-scored KRT18P10 profile across patient tissues and cancer cell-line models. KRT18P10 expression is associated with patient survival in 22 of 34 cancer types, with the highest sampling consensus in LUAD. Among the 18 cancer types available for tumor–normal comparison, KRT18P10 is differentially expressed in 14, with the highest sampling consensus in COAD. Additionally, KRT18P10 RNA expression shows 14,124 significant gene co-expression associations, with the highest sampling consensus in TGCT. Together, these results highlight LUAD, COAD, and TGCT as cancer lineages where KRT18P10 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for KRT18P10 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes KRT18P10 survival associations across molecular data types. KRT18P10 RNA expression shows survival associations in the most cancer types (22). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible KRT18P10 RNA expression–survival associations across cancer types. High KRT18P10 expression shows unfavorable associations in LUAD, PAAD, PCPG and ESCA, but favorable associations in THCA and BLCA. The LUAD Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify LUAD as the clearest survival context for KRT18P10 RNA expression.
This table summarizes KRT18P10 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 14. The strongest signals are observed in LUAD for RNA.
This table ranks reproducible tumor–normal expression differences for KRT18P10. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. KRT18P10 shows higher tumor expression in COAD, LUAD, KIRP, STAD, KIRC and BRCA. The COAD box plot shows higher KRT18P10 RNA expression in tumor versus normal tissue (log2 FC = +1.427, t-test p < 0.001).
This table shows molecular features associated with KRT18P10 in patient tissues and cancer cell lines. In patient samples, KRT18P10 shows the broadest associations at the RNA and protein expression levels, with TGCT recurring as the lineage with the largest associated feature set.
