KCNMA1-AS1

Across TCGA pan-cancer cohorts, KCNMA1-AS1 RNA differs between tumor and matched normal tissue in 6 of 18 cancer types tested, making tumor–normal expression one of KCNMA1-AS1’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where KCNMA1-AS1 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types KCNMA1-AS1 is over-expressed in tumor, although a few such as COAD and STAD show the opposite, repressed pattern.

KIRC, COAD, and STAD are the cancer types where KCNMA1-AS1 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in KCNMA1-AS1 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	Female	All	+0.098	<.001	11	view →
COAD	Female	All	−0.037	<.001	8	view →
STAD	All	All	−0.071	.022	2	view →
READ	Male	All	−0.033	.034	2	view →
KIRP	All	All	+0.016	.011	2	view →
CHOL	All	All	+0.117	.039	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 6 strongest of 6 lineages.

KCNMA1-AS1–KIRC

Open the KIRC breakdown →