heat shock protein family E (Hsp10) member 1 pseudogene 23Genealiases: []
Q-omics provides the consensus-scored HSPE1P23 profile across patient tissues and cancer cell-line models. HSPE1P23 expression is associated with patient survival in 10 of 34 cancer types, with the highest sampling consensus in THYM. Among the 18 cancer types available for tumor–normal comparison, HSPE1P23 is differentially expressed in 2, with the highest sampling consensus in LUAD. Additionally, HSPE1P23 RNA expression shows 6,009 significant pathway-activity associations, with the highest sampling consensus in STAD. Together, these results highlight THYM, LUAD, and STAD as cancer lineages where HSPE1P23 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for HSPE1P23 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes HSPE1P23 survival associations across molecular data types. HSPE1P23 RNA expression shows survival associations in the most cancer types (10). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible HSPE1P23 RNA expression–survival associations across cancer types. High HSPE1P23 expression shows unfavorable associations in THYM, UCEC, ACC, KIRC, THCA and STAD. The THYM Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify THYM as the clearest survival context for HSPE1P23 RNA expression.
This table summarizes HSPE1P23 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 2. The strongest signals are observed in LUAD for RNA.
This table ranks reproducible tumor–normal expression differences for HSPE1P23. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. HSPE1P23 shows lower tumor expression in LUAD and LUSC. The LUAD box plot shows higher HSPE1P23 RNA expression in normal versus tumor tissue (log2 FC = −0.164, t-test p = .001).
This table shows molecular features associated with HSPE1P23 in patient tissues and cancer cell lines. In patient samples, HSPE1P23 shows the broadest associations at the RNA and protein expression levels, with STAD recurring as the lineage with the largest associated feature set.
