heat shock protein family E (Hsp10) member 1 pseudogene 10Genealiases: []
Q-omics provides the consensus-scored HSPE1P10 profile across patient tissues and cancer cell-line models. HSPE1P10 expression is associated with patient survival in 20 of 34 cancer types, with the highest sampling consensus in KIRC. Among the 18 cancer types available for tumor–normal comparison, HSPE1P10 is differentially expressed in 4, with the highest sampling consensus in HNSC. Additionally, HSPE1P10 RNA expression shows 6,551 significant pathway-activity associations, with the highest sampling consensus in STAD. Together, these results highlight KIRC, HNSC, and STAD as cancer lineages where HSPE1P10 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for HSPE1P10 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes HSPE1P10 survival associations across molecular data types. HSPE1P10 RNA expression shows survival associations in the most cancer types (20). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible HSPE1P10 RNA expression–survival associations across cancer types. High HSPE1P10 expression shows unfavorable associations in KIRC, KIRP, ESCA and BRCA, but favorable associations in HNSC and CHOL. The KIRC Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify KIRC as the clearest survival context for HSPE1P10 RNA expression.
This table summarizes HSPE1P10 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 4. The strongest signals are observed in HNSC for RNA.
This table ranks reproducible tumor–normal expression differences for HSPE1P10. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. HSPE1P10 shows higher tumor expression in HNSC, COAD, LUSC and BLCA. The HNSC box plot shows higher HSPE1P10 RNA expression in tumor versus normal tissue (log2 FC = +0.043, t-test p = .016).
This table shows molecular features associated with HSPE1P10 in patient tissues and cancer cell lines. In patient samples, HSPE1P10 shows the broadest associations at the RNA and protein expression levels, with STAD recurring as the lineage with the largest associated feature set.
