GCNT1P3

Across TCGA pan-cancer cohorts, GCNT1P3 RNA differs between tumor and matched normal tissue in 10 of 18 cancer types tested, making tumor–normal expression one of GCNT1P3’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where GCNT1P3 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types GCNT1P3 is over-expressed in tumor, although a few such as LIHC and CHOL show the opposite, repressed pattern.

KIRC, HNSC, and THCA are the cancer types where GCNT1P3 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in GCNT1P3 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	All	All	+0.071	<.001	8	view →
HNSC	All	II,III,IV	+0.165	.001	7	view →
THCA	All	All	+0.109	<.001	7	view →
LIHC	All	All	−0.223	<.001	6	view →
LUSC	All	All	+0.168	.008	2	view →
BRCA	Female	All	+0.067	.044	2	view →
COAD	All	II,III,IV	+0.048	.030	2	view →
CHOL	All	All	−0.451	.028	1	view →
LUAD	All	All	+0.210	.036	1	view →
KICH	Female	All	−0.054	.044	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 10 strongest of 10 lineages.

GCNT1P3–KIRC

Open the KIRC breakdown →