FSIP2-AS1

Across TCGA pan-cancer cohorts, FSIP2-AS1 RNA differs between tumor and matched normal tissue in 13 of 18 cancer types tested, making tumor–normal expression one of FSIP2-AS1’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where FSIP2-AS1 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types FSIP2-AS1 is over-expressed in tumor, although a few such as BRCA and KICH show the opposite, repressed pattern.

KIRC, HNSC, and STAD are the cancer types where FSIP2-AS1 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in FSIP2-AS1 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	All	II,III,IV	+0.193	<.001	11	view →
HNSC	All	II,III,IV	+0.231	<.001	10	view →
STAD	All	II,III,IV	+0.449	<.001	7	view →
LUSC	All	All	+0.532	<.001	6	view →
BRCA	Female	All	−0.280	<.001	6	view →
KICH	All	All	−0.258	<.001	6	view →
LIHC	Male	All	+0.110	.002	4	view →
READ	Female	All	+0.752	<.001	3	view →
BLCA	All	III,IV	+0.573	.027	3	view →
COAD	All	All	+0.314	.001	3	view →
PRAD	All	All	+0.370	<.001	2	view →
LUAD	All	All	+0.255	.022	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 12 strongest of 13 lineages.

FSIP2-AS1–KIRC

Open the KIRC breakdown →