Across TCGA pan-cancer cohorts, FAM86DP Mutation is linked to patient survival in 2 of 34 cancer types, making it a survival-associated FAM86DP data layer compared with 23 for mass-spec protein.
The strongest signal is observed in prostate adenocarcinoma (PRAD), where higher FAM86DP Mutation is associated with worse disease-free survival. In most high-consensus cancer types, elevated FAM86DP expression acts as an unfavorable survival marker, although some lineages such as UCEC show a favorable association.
PRAD and UCEC are the cancer types where FAM86DP Mutation most reproducibly stratifies survival.