Across TCGA pan-cancer cohorts, EVI2A Mutation is linked to patient survival in 2 of 34 cancer types, making it a survival-associated EVI2A data layer compared with 22 for mass-spec protein and 2 for mass-spec protein.
The strongest signal is observed in prostate adenocarcinoma (PRAD), where higher EVI2A Mutation is associated with worse disease-free survival. In most high-consensus cancer types, elevated EVI2A expression acts as an unfavorable survival marker.
PRAD and STAD are the cancer types where EVI2A Mutation most reproducibly stratifies survival.