CYP3A52P

Across TCGA pan-cancer cohorts, CYP3A52P RNA differs between tumor and matched normal tissue in 2 of 18 cancer types tested, making tumor–normal expression one of CYP3A52P’s most consistent transcriptional readouts.

The strongest signal is observed in cholangiocarcinoma (CHOL), where CYP3A52P RNA is repressed in tumor relative to normal tissue. In most cancer types CYP3A52P is over-expressed in tumor, although a few such as CHOL show the opposite, repressed pattern.

CHOL and KIRC are the cancer types where CYP3A52P tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in CYP3A52P RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
CHOL	All	II,III,IV	−0.804	.004	2	view →
KIRC	Male	All	+0.279	.002	2	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 2 strongest of 2 lineages.

CYP3A52P–CHOL

Open the CHOL breakdown →