BNIP3P32

Across TCGA pan-cancer cohorts, BNIP3P32 RNA differs between tumor and matched normal tissue in 5 of 18 cancer types tested, making tumor–normal expression one of BNIP3P32’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal papillary cell carcinoma (KIRP), where BNIP3P32 RNA is repressed in tumor relative to normal tissue. In most cancer types BNIP3P32 is over-expressed in tumor, although a few such as KIRP and KIRC show the opposite, repressed pattern.

KIRP, KIRC, and THCA are the cancer types where BNIP3P32 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in BNIP3P32 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRP	Male	All	−0.250	.001	8	view →
KIRC	Male	All	−0.145	<.001	7	view →
THCA	All	All	−0.149	.001	2	view →
KICH	All	All	−0.122	.009	2	view →
LUSC	All	II,III,IV	−0.026	.038	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 5 strongest of 5 lineages.

BNIP3P32–KIRP

Open the KIRP breakdown →