BCL2 interacting protein 3 pseudogene 32Genealiases: []
Q-omics provides the consensus-scored BNIP3P32 profile across patient tissues and cancer cell-line models. BNIP3P32 expression is associated with patient survival in 17 of 34 cancer types, with the highest sampling consensus in KICH. Among the 18 cancer types available for tumor–normal comparison, BNIP3P32 is differentially expressed in 5, with the highest sampling consensus in KIRP. Additionally, BNIP3P32 RNA expression shows 12,762 significant protein co-abundance associations, with the highest sampling consensus in GBM. Together, these results highlight KICH, KIRP, and GBM as cancer lineages where BNIP3P32 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for BNIP3P32 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes BNIP3P32 survival associations across molecular data types. BNIP3P32 RNA expression shows survival associations in the most cancer types (17). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible BNIP3P32 RNA expression–survival associations across cancer types. High BNIP3P32 expression shows unfavorable associations in LUSC, COAD, TGCT and LIHC, but favorable associations in KICH and MESO. The KICH Kaplan–Meier curve shows clear separation, with the low-expression group declining faster, consistent with the favorable association (log-rank p = .005). Together, the overview and detailed table identify KICH as the clearest survival context for BNIP3P32 RNA expression.
This table summarizes BNIP3P32 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 5. The strongest signals are observed in KIRP for RNA.
This table ranks reproducible tumor–normal expression differences for BNIP3P32. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. BNIP3P32 shows lower tumor expression in KIRP, KIRC, THCA, KICH and LUSC. The KIRP box plot shows higher BNIP3P32 RNA expression in normal versus tumor tissue (log2 FC = −0.250, t-test p = .001).
This table shows molecular features associated with BNIP3P32 in patient tissues and cancer cell lines. In patient samples, BNIP3P32 shows the broadest associations at the RNA and protein expression levels, with GBM recurring as the lineage with the largest associated feature set.
