Q-omics provides the consensus-scored BMS1P13 profile across patient tissues and cancer cell-line models. BMS1P13 expression is associated with patient survival in 11 of 34 cancer types, with the highest sampling consensus in KICH. Among the 18 cancer types available for tumor–normal comparison, BMS1P13 is differentially expressed in 3, with the highest sampling consensus in HNSC. Additionally, BMS1P13 RNA expression shows 3,551 significant pathway-activity associations, with the highest sampling consensus in STAD. Together, these results highlight KICH, HNSC, and STAD as cancer lineages where BMS1P13 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for BMS1P13 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes BMS1P13 survival associations across molecular data types. BMS1P13 RNA expression shows survival associations in the most cancer types (11). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible BMS1P13 RNA expression–survival associations across cancer types. High BMS1P13 expression shows unfavorable associations in KICH, LIHC, KIRC, LUAD and MESO, but favorable associations in SKCM. The KICH Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify KICH as the clearest survival context for BMS1P13 RNA expression.
This table summarizes BMS1P13 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 3. The strongest signals are observed in BRCA for RNA.
This table ranks reproducible tumor–normal expression differences for BMS1P13. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. BMS1P13 shows lower tumor expression in HNSC, BRCA and KIRC. The HNSC box plot shows higher BMS1P13 RNA expression in normal versus tumor tissue (log2 FC = −0.039, t-test p = .021).
This table shows molecular features associated with BMS1P13 in patient tissues and cancer cell lines. In patient samples, BMS1P13 shows the broadest associations at the RNA and protein expression levels, with STAD recurring as the lineage with the largest associated feature set.
