ARMC10P1

Across TCGA pan-cancer cohorts, ARMC10P1 RNA differs between tumor and matched normal tissue in 12 of 18 cancer types tested, making tumor–normal expression one of ARMC10P1’s most consistent transcriptional readouts.

The strongest signal is observed in colon adenocarcinoma (COAD), where ARMC10P1 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types ARMC10P1 is over-expressed in tumor, although a few such as THCA and KICH show the opposite, repressed pattern.

COAD, THCA, and BRCA are the cancer types where ARMC10P1 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in ARMC10P1 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
COAD	Female	III,IV	+0.650	<.001	9	view →
THCA	Female	III,IV	−0.190	.017	5	view →
BRCA	All	All	+0.124	<.001	4	view →
HNSC	All	All	+0.121	.002	4	view →
KICH	All	All	−0.219	<.001	3	view →
LUAD	All	All	+0.200	.002	3	view →
READ	All	III,IV	+0.440	.002	2	view →
CHOL	All	All	+0.226	.001	2	view →
LUSC	All	All	+0.111	.011	2	view →
LIHC	All	All	+0.056	.016	2	view →
STAD	All	II,III,IV	+0.237	.027	1	view →
BLCA	All	IV	−0.120	.036	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 12 strongest of 12 lineages.

ARMC10P1–COAD

Open the COAD breakdown →