ANKRD30A

Across TCGA pan-cancer cohorts, ANKRD30A RNA differs between tumor and matched normal tissue in 7 of 18 cancer types tested, making tumor–normal expression one of ANKRD30A’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where ANKRD30A RNA is repressed in tumor relative to normal tissue. In most cancer types ANKRD30A is over-expressed in tumor, although a few such as KIRC and THCA show the opposite, repressed pattern.

KIRC, THCA, and KICH are the cancer types where ANKRD30A tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in ANKRD30A RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	Male	II,III,IV	−0.082	<.001	11	view →
THCA	All	III,IV	−0.054	<.001	8	view →
KICH	All	II,III,IV	+0.838	<.001	6	view →
COAD	All	All	+0.017	.006	6	view →
LIHC	All	All	+0.029	.013	5	view →
HNSC	All	IV	+0.007	.036	4	view →
BLCA	Male	III,IV	+0.037	.025	2	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 7 strongest of 7 lineages.

ANKRD30A–KIRC

Open the KIRC breakdown →