ANKRD26P1

Mutation & survival
SurvivalMutationKaplan–Meier · TCGA cohorts

Across TCGA pan-cancer cohorts, ANKRD26P1 Mutation is linked to patient survival in 3 of 34 cancer types, making it a survival-associated ANKRD26P1 data layer compared with 20 for mass-spec protein.

The strongest signal is observed in mesothelioma (MESO), where higher ANKRD26P1 Mutation is associated with worse overall survival. In most high-consensus cancer types, elevated ANKRD26P1 expression acts as an unfavorable survival marker.

MESO, STAD, and UCEC are the cancer types where ANKRD26P1 Mutation most reproducibly stratifies survival.

Mutation survival associations by lineage

Ranked by sampling consensus. AUC1 and AUC2 indicate survival in the high- and low-expression groups, respectively; the lower AUC marks the poorer-surviving group. p-values are from the log-rank test.
LineageMeasureSplitStageAUC1
high
AUC2
low
pSampling consensus
MESOOSMedianIII,IV0.0770.580<.00142view →
STADOSMedianII,III,IV0.0030.677<.00130view →
UCECOSMedianIV0.2310.592.0366view →
Pink = unfavorable, green = favorable. Showing the 3 strongest of 3 lineages.

ANKRD26P1–MESO (OS)

Kaplan–Meier survival curve for ANKRD26P1 mutant vs wild-type samples in MESO.

Open the MESO breakdown →

Exploration