Q-omics provides the consensus-scored AKIRIN1P2 profile across patient tissues and cancer cell-line models. AKIRIN1P2 expression is associated with patient survival in 18 of 34 cancer types, with the highest sampling consensus in KICH. Among the 18 cancer types available for tumor–normal comparison, AKIRIN1P2 is differentially expressed in 3, with the highest sampling consensus in KIRC. Additionally, AKIRIN1P2 RNA expression shows 12,181 significant protein co-abundance associations, with the highest sampling consensus in GBM. Together, these results highlight KICH, KIRC, and GBM as cancer lineages where AKIRIN1P2 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for AKIRIN1P2 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes AKIRIN1P2 survival associations across molecular data types. AKIRIN1P2 RNA expression shows survival associations in the most cancer types (18). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible AKIRIN1P2 RNA expression–survival associations across cancer types. High AKIRIN1P2 expression shows unfavorable associations in KICH, ACC, KIRC, LIHC and PAAD, but favorable associations in BLCA. The KICH Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify KICH as the clearest survival context for AKIRIN1P2 RNA expression.
This table summarizes AKIRIN1P2 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 3. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for AKIRIN1P2. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. AKIRIN1P2 shows lower tumor expression in KIRC and THCA and higher tumor expression in COAD. The KIRC box plot shows higher AKIRIN1P2 RNA expression in normal versus tumor tissue (log2 FC = −0.024, t-test p = .016).
This table shows molecular features associated with AKIRIN1P2 in patient tissues and cancer cell lines. In patient samples, AKIRIN1P2 shows the broadest associations at the RNA and protein expression levels, with GBM recurring as the lineage with the largest associated feature set.
