Across TCGA pan-cancer cohorts, VPS26CP1 RNA differs between tumor and matched normal tissue in 1 of 18 cancer types tested, making tumor–normal expression one of VPS26CP1’s most consistent transcriptional readouts.
The strongest signal is observed in prostate adenocarcinoma (PRAD), where VPS26CP1 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types VPS26CP1 is over-expressed in tumor.
PRAD are the cancer types where VPS26CP1 tumor–normal differential expression is most reproducible.