VN1R87P

Across TCGA pan-cancer cohorts, VN1R87P RNA differs between tumor and matched normal tissue in 4 of 18 cancer types tested, making tumor–normal expression one of VN1R87P’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal clear cell carcinoma (KIRC), where VN1R87P RNA is repressed in tumor relative to normal tissue. In most cancer types VN1R87P is over-expressed in tumor, although a few such as KIRC and KIRP show the opposite, repressed pattern.

KIRC, KIRP, and THCA are the cancer types where VN1R87P tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in VN1R87P RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRC	Male	All	−0.151	<.001	12	view →
KIRP	Male	All	−0.129	<.001	6	view →
THCA	All	All	+0.111	.001	4	view →
LIHC	All	All	+0.192	.002	3	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 4 strongest of 4 lineages.

VN1R87P–KIRC

Open the KIRC breakdown →