VN1R78P

Across TCGA pan-cancer cohorts, VN1R78P RNA differs between tumor and matched normal tissue in 4 of 18 cancer types tested, making tumor–normal expression one of VN1R78P’s most consistent transcriptional readouts.

The strongest signal is observed in liver hepatocellular carcinoma (LIHC), where VN1R78P RNA is more highly expressed in tumor relative to normal tissue. In most cancer types VN1R78P is over-expressed in tumor, although a few such as THCA and KICH show the opposite, repressed pattern.

LIHC, KIRC, and THCA are the cancer types where VN1R78P tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in VN1R78P RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
LIHC	All	All	+0.072	.012	2	view →
KIRC	All	All	+0.050	.013	2	view →
THCA	Female	All	−0.032	.046	1	view →
KICH	All	All	−0.030	.019	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 4 strongest of 4 lineages.

VN1R78P–LIHC

Open the LIHC breakdown →