VN1R48P

Across TCGA pan-cancer cohorts, VN1R48P RNA differs between tumor and matched normal tissue in 11 of 18 cancer types tested, making tumor–normal expression one of VN1R48P’s most consistent transcriptional readouts.

The strongest signal is observed in thyroid carcinoma (THCA), where VN1R48P RNA is repressed in tumor relative to normal tissue. In most cancer types VN1R48P is over-expressed in tumor, although a few such as THCA and KIRC show the opposite, repressed pattern.

THCA, KIRC, and BRCA are the cancer types where VN1R48P tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in VN1R48P RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
THCA	Female	II,III,IV	−0.742	<.001	11	view →
KIRC	Female	All	−0.126	<.001	11	view →
BRCA	All	III,IV	+0.911	<.001	6	view →
KICH	All	All	−0.124	<.001	6	view →
LIHC	All	All	+0.187	.013	4	view →
HNSC	All	II,III,IV	−0.014	.043	4	view →
CHOL	All	All	+0.832	.004	2	view →
LUAD	All	All	+0.124	.014	2	view →
COAD	All	II,III,IV	+0.055	.024	2	view →
LUSC	All	All	−0.052	.004	2	view →
KIRP	Male	II,III,IV	−0.067	.023	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 11 strongest of 11 lineages.

VN1R48P–THCA

Open the THCA breakdown →